Ethan Dmitrovsky, MD, Oral History Interview, March 3, 2015

Ethan Dmitrovsky, MD, Oral History Interview, March 3, 2015

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Description

Major Topics Covered:

  • Personal and educational background; overseas experience and inspiration
  • Research: cell differentiation; retinoids in leukemic differentiation; lung cancer studies
  • The Role of the Provost; personal goals; institutional mandate; detailed of plans
  • MD Anderson’s ten-year strategic plan
  • Research in an environment of changing economics and health care delivery

Identifier

DmitrovskyE_01_20150303

Publication Date

3-5-2015

Publisher

The Making Cancer History® Voices Oral History Collection, The University of Texas MD Anderson Cancer Center

City

Houston, Texas

Creative Commons License

Creative Commons Attribution-Noncommercial-No Derivative Works 3.0 License
This work is licensed under a Creative Commons Attribution-Noncommercial-No Derivative Works 3.0 License.

Topics Covered

University of Texas MD Anderson Cancer Center; UT MD Anderson Cancer Center; University of Texas System. M.D. Anderson Cancer Center; M.D. Anderson Hospital and Tumor Institute at Houston; University of Texas M.D. Anderson Cancer Center; M.D. Anderson Hospital and Tumor Institute

Disciplines

History of Science, Technology, and Medicine | Oncology | Oral History

About the Interview

About the Interview Subject:

Ethan Dmitrovsky, MD came to MD Anderson in 2013 to serve as the institution’s Provost and Executive Vice President. He has a faculty appointment in the Department of Thoracic/Head and Neck Medical Oncology in the Division of Cancer Medicine.

Dr. Dmitrovsky’s translational research areas include: retinoid differentiation-based therapy for acute promyelocytic leukemia (APL); developed the molecular genetic test used to detect the PML/RARalpha transcript from the APL t(15;17) rearrangement; retinoid mechanisms leading to cell cycle arrest and repair of DNA damage in normal/malignant lung epithelial cells; engineered transgenic mouse models that express wild-type or proteasomal degradation-resistant cyclin E species in the lung; derived lung cancer cell lines leading to a new model to assess activity of lung cancer therapy and chemopreventive agents (antineoplastics).

Conditions Governing Access

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Ethan Dmitrovsky, MD, Oral History Interview, March 3, 2015

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