Inhibition of p38 kinase in cancer cells with Hybrid EMT induces Sensitivity to TGF-β Inhibitor And Disrupts Plasticity & Stemness

Authors

Kelsey Margarita Medina, The University of Texas Health Science Center at Houston McGovern Medical SchoolFollow
Micaela Morgado, The University of Texas MD Anderson Cancer Center
Petra Den Hollander, The University of Texas MD Anderson Cancer Center
Sendurai A. Mani, The University of Texas MD Anderson Cancer Center

Files

Publisher

The University of Texas MD Anderson Cancer Center

City

Houston

Keywords

Breast Cancer, Epithelial to Mesenchymal Transition (EMT), cell plasticity, p38 Inhibitor (SB203580), FOXC2

Creative Commons License

This work is licensed under a Creative Commons Attribution-Noncommercial-No Derivative Works 3.0 License.

Program Affiliation

Partnership for Careers in Cancer Science and Medicine

Recommended Citation

Medina, Kelsey Margarita; Morgado, Micaela; Den Hollander, Petra; and Mani, Sendurai A., "Inhibition of p38 kinase in cancer cells with Hybrid EMT induces Sensitivity to TGF-β Inhibitor And Disrupts Plasticity & Stemness" (2021). Summer Experience 2021. 114.
https://openworks.mdanderson.org/sumexp21/114